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Several studies have shown that DPP4-i can cross the blood-brain barrier, allowing them to act directly within the brain, and influence neuroinflammatory processes, the aggregation of pathological proteins, and neurodegeneration ( Kosaraju et al
The key is to use the lowest effective volume and frequency to correct the deficiency or meet the metabolic need, and to adjust as the patients condition improves
In apoptosis, mitochondrial outer membrane permeabilization (MOMP) is the point of no return
Pernicious anaemia and deficiency with neurological involvement require more intensive treatment with alternate-day injections until improvement, followed by lifelong maintenance
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