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1ce), mirroring the previously reported phenotypes of germline global Gipr KO mice and Gipr fx/fx mice crossed with Nestin-Cre to drive KO throughout the central nervous system (CNS) 4,9
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However, this work once again highlights the remarkable metabolic plasticity of tumour cellsa key aspect of cancer biologywhich allows them to adapt to adverse nutritional conditions and, in many cases, to develop therapy resistance
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