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The American Journal of Clinical Nutrition, 72(2), 618S623S
Taken together, these observations suggest that except in vitamin B 12 deficiency (which only responds to cyanocobalamin treatment because of the inactivation of methionine synthase), folate treatment should be the most adequate therapy to reverse hyperhomocysteinemia detected both under basal conditions and after an oral methionine load
CJC-1295 is a GHRH analog
